Path: utzoo!utgpu!news-server.csri.toronto.edu!cs.utexas.edu!usc!ucla-cs!news From: Tom.Tempe@p0.f101.n114.z1.fidonet.org (Tom Tempe) Newsgroups: sci.med.aids Subject: symbiosis? Message-ID: <1991Jan28.120515.22578@cs.ucla.edu> Date: 28 Jan 91 02:41:37 GMT Sender: ufgate@stjhmc.fidonet.org (newsout1.26) Organization: FidoNet node 1:114/101.0 - The Land of Oz BBS, Phoenix AZ Lines: 20 Approved: phil@wubios.wustl.edu Note: non-commercial reproduction. Nntp-Posting-Host: squid.cs.ucla.edu Archive-Number: 2944 I've been wondering for a while if some aspects of HIV ( high mutation rate, ability to hide from immune system, variability) could be due to a symbiotic relationship with a prion. I don't have a degree so getting research done is virtually impossible. If there is a prion based on part of a mitochondra ( unknown but not impossible, per Dr. Prusiner) it would show universal selfness and , if present in sufficient quantity could hide other agents present from the immune system. If it had a zinc finger instead of an unidentified sugar molecule (again, unknown but possible, per Dr. Prusiner) it could be mutagenic ( possible but not known, per Alan Wilson). Since it's the type of quasi-organism that wouldn't be noticed unless it was checked for specifically it could slip past current research. Is there any research on this , or if not a good way to get it researched (categorizing patients with different immune responses and rates of symptom onset and looking for it by electrophoresis and looking for zinc) so a determination could be made? -- Uucp: ...{gatech,ames,rutgers}!ncar!asuvax!stjhmc!101.0!Tom.Tempe Internet: Tom.Tempe@p0.f101.n114.z1.fidonet.org