Relay-Version: version B 2.10 5/3/83; site utzoo.UUCP Posting-Version: version B 2.10.1 6/24/83; site ecsvax.UUCP Path: utzoo!watmath!clyde!bonnie!akgua!mcnc!ecsvax!emigh From: emigh@ecsvax.UUCP (Ted Emigh) Newsgroups: net.motss,net.bio Subject: Re: gay genes Message-ID: <1299@ecsvax.UUCP> Date: Mon, 20-May-85 10:02:16 EDT Article-I.D.: ecsvax.1299 Posted: Mon May 20 10:02:16 1985 Date-Received: Wed, 22-May-85 02:13:16 EDT References: <4086@allegra.UUCP> <1901@ut-sally.UUCP> <452@psivax.UUCP> Reply-To: emigh@ecsvax.UUCP (Ted Emigh) Organization: NC State University Lines: 17 Keywords: Sickle Cell Anemia; Thalassemia Major Xref: watmath net.motss:1712 net.bio:205 Summary: In article <452@psivax.UUCP> friesen@psivax.UUCP (Stanley friesen) writes: > Actually, the heterozygote(only one copy) sickle cell >advantage is restricted to tropical Africa, and it is due to >immunity to African Sleeping Sickness not malaria. Actually, the heterozygote sickle cell advantage is to malaria (Plasmodium falciparum). While the major occurence of sickle cell anemia gene is in Central Africa, it is also found in other malarial environments (Persian Gulf, Central India, etc). Another abnormality also is associated with malaria -- Thalassemia major, which occurs in relatively high frequencies in the Mediterranean countries (also where malaria had a high incidence). -- Ted H. Emigh Genetics and Statistics, North Carolina State U, Raleigh NC USENET: {akgua decvax duke ihnp4 unc}!mcnc!ecsvax!emigh ARPA: decvax!mcnc!ecsvax!emigh@BERKELEY