Relay-Version: version B 2.10 5/3/83; site utzoo.UUCP Posting-Version: version B 2.10.1 (Denver Mods 7/26/84) 6/24/83; site drutx.UUCP Path: utzoo!watmath!clyde!cbosgd!cbdkc1!desoto!cord!pierce!bentley!hoxna!houxm!mtuxo!drutx!jca From: jca@drutx.UUCP (ArnsonJC) Newsgroups: net.bio,net.med Subject: Re: Malaria and sickle cell Message-ID: <306@drutx.UUCP> Date: Mon, 26-Aug-85 19:34:25 EDT Article-I.D.: drutx.306 Posted: Mon Aug 26 19:34:25 1985 Date-Received: Wed, 28-Aug-85 20:29:42 EDT References: <191@tekig5.UUCP> <314@kitty.UUCP> <678@cybvax0.UUCP> Organization: AT&T Information Systems Laboratories, Denver Lines: 7 Xref: watmath net.bio:252 net.med:2214 It is not the HgbS that is responsible for malaria "immunity". It is due to lack of the Duffy Blood Group System genes, both a and b. [ie. Fya-, Fyb-]. It just happens that both the HgbS genes and Duffy {or should I say lack of} genes are both found in the same segment of the population. {Blacks} jill